Cov paj hlwb tsis muaj zog tuaj yeem ua raulub plawv dhia qis, lossis bradycardia. Cov tib neeg uas muaj cov hlab ntsha tsis muaj zog ntau dhau uas ua rau lub plawv dhia qis kuj tseem tuaj yeem ua rau muaj kev pheej hmoo rau thawj-degree lub plawv nres.
Vim li cas vagal stimulation txo lub plawv dhia?
Ob ceg ntawm lub paj hlwb autonomic ua haujlwm ua ke kom nce lossis qeeb lub plawv dhia. Cov hlab ntsha vagus ua rau ntawm lub sinoatrial node, slows nws conduction thiab modulating vagal tone, ntawm lub neurotransmitter acetylcholine thiab downstream hloovrau ionic tam sim no thiab calcium ntawm lub plawv hlwb.
Qhov cuam tshuam ntawm vagal stimulation yog dab tsi?
Kev tawm dag zog los ntawm sab laug mid-cervical vagus nerve feem ntau ua rau suab hloov pauv, hnoos, dyspnea, dysphagia, thiab mob caj dab lossis paresthesias. Sab laug lub ncauj tsev menyuam VNS ntseeg tau tias yuav txo qis cov teebmeem plawv xws li bradycardia lossis asystole (feem ntau yog kho los ntawm txoj cai vagus paj hlwb).
Puas vagus paj stimulation txo lub plawv dhia?
Zoo. VNS tuaj yeem siv tau zoo thiab sai kom txo lub plawv dhia, hauv cov chaw mob hnyav, thaum txuas nrog rau sab nraud pacing system.
Cov paj hlwb vagus ua rau bradycardia li cas?
Nco ntsoov, cov hlab ntsha vagus txhawb qee cov leeg hauv plawv uas pab ua kom lub plawv dhia qeeb. Thaum nws overreacts, nws tuaj yeem ua rau poob siab sai hauv lub plawv dhia thiab ntshav siab, ua raufainting.
