Txog 20% ntawm cov neeg mob nrog hypercalcemia tsim polyuria. Cov txheej txheem postulated yog downregulation ntawm aquaporin-2 dej raws, thiab calcium deposition nyob rau hauv lub medulla nrog ob tug tubulointerstitial raug mob, ua rau impaired tiam ntawm interstitial osmotic gradient.
Puas muaj calcium ntau ua rau tso zis ntau zaus?
Cov calcium ntau dhau ua rau koj lub raum ua haujlwm hnyav los lim nws. Qhov no tuaj yeem ua rau nqhis dej ntau dhau thiab tso zis ntau zaus. digestive system. Hypercalcemia tuaj yeem ua rau mob plab, xeev siab, ntuav thiab cem quav.
Hypercalcemia ua rau nephrogenic DI li cas?
Hypercalcemia induces targeted autophagic degradation of aquaporin-2 ntawm qhov pib ntawm nephrogenic diabetes insipidus.
Yuav ua li cas hypercalcemia cuam tshuam cov zis concentration?
Hypercalcemia nce tso zis ntawm iCa, iMg, sodium, phosphate, potassium, thiab chloride; tso zis ntau ntxiv; thiab txo cov zis osmolality thiab lub ntiajteb txawj nqus. Kev tswj hwm dextrose nce ntshav cov tshuaj insulin; txo iMg, potassium, thiab phosphate concentrations; thiab txo qis tso zis ntawm iMg.
Vim li cas hypercalcemia ua rau osmotic diuresis?
Ib qho osmotic diuresis kuj tuaj yeem tshwm sim los ntawm kev tsim cov urea ntau dhau vim yog kev tswj hwm cov protein ntau. Hypercalcemia tshuaj lom distal tubular muaj nuj nqi, ua rau ntau dhau ntawm dilutetso zis.
