Cholecystokinin (CCK) tau tshawb pom hauv xyoo 1928 hauv jejunal extracts raws li qhov ua rau lub gallbladder contraction. Tom qab ntawd nws tau pom tias yog cov tswv cuab ntawm tsev neeg peptide, uas yog tag nrho cov ligands rau CCK1 thiab CCK2 receptors. CCK peptides paub tias yuav tsum tau ua ke nyob rau hauv cov hnyuv me endocrine I-cells thiab cerebral neurons.
Yuav ua li cas thiaj ua rau lub gallbladder cog lus?
Cholecystokinin (CCK) yog tso tawm los ntawm mucosal endocrine cells nyob rau hauv txoj hnyuv me nyob rau hauv cov lus teb rau pluas mov [1], thiab nws yog classical paub los txhawb lub gallbladder contraction.
Cov tshuaj plab hnyuv dab tsi ua rau lub zais zis contraction?
Gallbladder contractions yog induced los ntawm kev ua ntawm cholecystokinin (CCK), peptide hormone tso tawm los ntawm cov neuroendocrine hlwb ntawm cov hnyuv me, ntawm CCK-A receptors ntawm interstitial Cell ntawm Cajal.
Yuav ua li cas cov tshuaj hormones txhawb kev tso cov kua tsib ntawm lub gallbladder?
Thaum txhawb nqa los ntawm hormone cholecystokinin (CCK), lub gallbladder cog lus, thawb cov kua tsib los ntawm cystic duct thiab mus rau hauv cov kua tsib.
Puas zais zais zais zais zais zais cia?
Secretin txhawb nqa cov kua tsib ntawm lub siab mus rau lub gallbladder. CCK txhawb lub gallbladder kom cog lus, ua rau cov kua tsibzais cia rau hauv duodenum, raws li qhia hauv qab no.