Piv txwv li, qhov siab, siab tshaj T nthwv dej hauv tus neeg mob uas tsis tau peb qhov kev lim ntshav zoo li yuav sawv cev hyperkalemia, thaum siab "hyperacute" T yoj hauv tus neeg mob yws ntawm Qhov pib mob ntawm kev sib tsoo, mob hauv siab hauv siab tuaj yeem sawv cev rau qhov pib mob ntawm kev hloov pauv myocardial ischemia.
T yoj peaked qhia li cas?
Nov thiab siab peaked T yoj (A) yog ib qho kos npe ntxov ntawm hyperkalemia. Nws yog qhov txawv txav rau T waves siab dua 5 mm nyob rau hauv limb leads thiab siab tshaj 10 mm nyob rau hauv siab leads. Hyperkalemia yuav tsum xav tias yog tias cov kev txwv no dhau los ntawm ntau tshaj ib qho txhuas.
Yuav ua li cas koj kho cov peaked T waves?
Kev kho mob thawj zaug tseem ceeb tshaj plaws uas yuav tsum tau ua yog pom EKG hloov pauv yog kev tswj hwm calcium gluconate lossis calcium chloride. Qee cov kws kho mob thaum muaj xwm txheej ceev tau tawm tswv yim rau kev tswj hwm calcium nrog lub siab T-yoj ib leeg, thaum lwm tus tsuas yog kho yog tias pom pom ntxiv.
T yoj hauv ib txwm ECG qhia dab tsi?
T yoj ntawm ECG (T-ECG) sawv cev repolarization ntawm ventricular myocardium. Nws cov morphology thiab lub sijhawm feem ntau yog siv los kuaj xyuas cov kab mob pathology thiab ntsuas kev pheej hmoo ntawm kev phom sij txog lub neej ventricular arrhythmias.
Puas txawv txav T yoj txhais tau tias lub plawv nres?
tag nrho, 25% ntawm cov neeg mob muaj T-wave abnormality (flattening los yog ib qho degree ntawm inversion) ntawm thawj ECG. T-wave hloov tau txuam nrognrog muaj kev pheej hmoo ntau ntxiv rau qhov kawg ntawm kev tuag, myocardial infarction, rov ua dua, lossis kuaj pom tau zoo raws li tus kab mob coronary artery (CAD).
