Ib ob peb qhov ua rau prerenal AKI suav nrog tab sis tsis txwv rau;intravascular ntim depletion, hypotension, sepsis, poob siab, tshaj diuresis, plawv tsis ua hauj lwm, cirrhosis, ob sab lub raum hlab ntsha stenosis / tib lub raum ua haujlwm uas ua rau lub raum tsis zoo los ntawm angiotensin-hloov enzyme (ACE) inhibitors, thiab los ntawm lwm tus…
Qhov twg hauv qab no yog qhov ua rau Prerenal mob raum tsis ua haujlwm?
Cov tshuaj tseem ceeb uas ua rau lub raum tsis ua haujlwm prerenal yog angiotensin-hloov enzyme (ACE) inhibitors thiab nonsteroidal anti-inflammatory tshuaj (NSAIDs). Qhov inhibition ntawm ACE tiv thaiv kev hloov pauv ntawm angiotensin I mus rau angiotensin II, ua rau txo qis ntawm angiotensin II.
Yuav ua li cas ua rau ua ntej lub raum tsis ua haujlwm?
Intravascular volume depletion yog qhov ua rau ua ntej lub raum tsis ua haujlwm. Intravascular ntim depletion tuaj yeem yog qhov tshwm sim ntawm qhov ncauj tsis zoo lossis poob dej ntau dhau.
Anuric lub raum tsis ua haujlwm yog dab tsi?
Anuria lossis anuresis tshwm sim thaum lub raum tsis tsim cov zis. Ib tug neeg yuav xub ntsib oliguria, lossis tso zis tsawg, thiab tom qab ntawd nce mus rau anuria. Kev tso zis yog qhov tseem ceeb hauv kev tshem tawm cov khoom pov tseg thiab cov kua dej ntau dhau ntawm koj lub cev. Koj ob lub raum tsim tawm ntawm 1 mus rau 2 quarts ntawm cov zis ib hnub.
Dab tsi yog pathophysiology ntawm Prerenal tsis ua haujlwm?
Nyob rau hauv preren altsis ua haujlwm, GFR yog kev nyuaj siab los ntawm kev cuam tshuam rau lub raum perfusion. Tubular thiab glomerular muaj nuj nqi nyob li qub. Intrinsic raum tsis ua hauj lwm suav nrog cov kab mob ntawm lub raum nws tus kheej, feem ntau cuam tshuam rau glomerulus lossis tubule, uas cuam tshuam nrog kev tso tawm ntawm lub raum afferent vasoconstrictors.
