Steroids txhawb nqa cov piam thaj los ntawm lub siab thiab inhibit peripheral qabzib uptake , ua rau insulin tsis kam. Yog tias pancreas tsis tuaj yeem tsim cov tshuaj insulin txaus los them rov qab, hyperglycemia tuaj yeem tshwm sim.
Vim li cas cov tshuaj steroids cuam tshuam rau cov ntshav qab zib?
Yog tias koj muaj ntshav qab zib thiab noj tshuaj steroid, koj cov ntshav qabzib yuav nce ntxiv. Cov tshuaj steroid tuaj yeem nce qib ntshav qabzib los ntawm txo qis kev ua ntawm insulin(ua rau insulin tsis kam) thiab ua rau lub siab tso cov piam thaj rau hauv cov hlab ntsha.
Puas corticosteroids cuam tshuam cov ntshav qabzib?
Ib qho ntawm cov kev mob tshwm sim ntawm qhov ncauj corticosteroids yog qhov lawv tuaj yeem ua rau cov ntshav qabzib nce ntxivthiab nce insulin tsis kam, uas tuaj yeem ua rau mob ntshav qab zib hom 2.
Puas corticosteroids ua rau hypo lossis hyperglycemia?
Cov neeg mob feem ntau tau muab cov tshuaj glucocorticoids tsawg kawg yog sib npaug li 40 mg / hnub rau ntau tshaj 2 hnub txhim kho hyperglycemia [11]. Nws yog qhov paub zoo tias kev kho glucocorticoid tuaj yeem ua rau mob ntshav qab zib hom 2 tshiab (T2DM) thiab ua rau tsis zoo rau cov neeg mob ntshav qab zib uas muaj ntshav qab zib hom 2 [10].
Vim li cas hydrocortisone ua rau hyperglycemia?
Txawm li cas los xij, hydrocortisone yog ib qho muaj zog glucocorticoid thiab nws stimulates gluconeogenesis hauv ob lub siab thiab cov ntaub so ntswg peripheral. Nws yogQhov ua tau tias kev kho corticosteroid tuaj yeem ua rau hyperglycemia thiab qhov zaus ntawm kev siv cov tshuaj insulin tuaj yeem nce nrog corticosteroid raug [7].
